Usually, the loss in the heart’s pumping action is a symptom of an underlying heart problem.
Heart valve disease caused by past rheumatic fever or other infections
Infections of the heart valves and/or heart muscle (i.e., endocarditis)
Cardiac arrhythmias (irregular heartbeats)
Cardiomyopathy, or another primary disease of the heart muscle
Chronic lung disease
Anemia
High blood pressure (hypertension)
Hemorrhage (excessive bleeding)
CHF leads to breathlessness, fatigue, and accumulation of fluid in the lungs or the veins (primarily in the legs) or both.
People with CHF have insufficient oxygenation of the heart, which can damage the heart muscle. Such damage may be reduced by taking L-carnitine supplements.5 L-carnitine is a natural substance made from the amino acids, lysine and methionine. Levels of L-carnitine are low in people with CHF;6 therefore, many doctors recommend that those with CHF take 500 mg of L-carnitine two to three times per day.
Most L-carnitine/CHF research has used a modified form of the supplement called propionyl-L-carnitine (PC). In one double-blind trial, people using 500 mg of PC per day had a 26% increase in exercise capacity after six months.7 In double-blind research, other indices of heart function have also improved after taking 1 gram of PC twice per day.8 It remains unclear whether propionyl-L-carnitine has unique advantages over L-carnitine, as limited research in animals and humans has also shown very promising effects of the more common L-carnitine.9
Magnesium deficiency frequently occurs in people with CHF, and such a deficiency may lead to heart arrhythmias. Magnesium supplements have reduced the risk of these arrhythmias.10 People with CHF are often given drugs that deplete both magnesium and potassium; a deficiency of either of these minerals may lead to an arrhythmia.11 Many doctors suggest magnesium supplements of 300 mg per day.
Whole fruit and fruit and vegetable juice, which are high in potassium, are also recommended by some doctors. One study showed that elderly men who consumed food prepared with potassium-enriched salt (containing about half potassium chloride and half sodium chloride) had a 70% reduction in deaths due to heart failure and a significant reduction in medical costs for cardiovascular disease, when compared with men who continued to use regular salt.12 While increasing potassium intake can be beneficial for heart patients, this dietary change should be discussed with a healthcare provider, because several drugs given to people with CHF may actually cause retention of potassium, making dietary potassium, even from fruit, dangerous.
Albany Medical Center is the first healthcare institution in the region to introduce aquapheresis-an innovative therapy which removes dangerous levels of excess fluid in patients suffering from congestive heart failure. The therapy has proven to be more effective in removing excess fluid than standard treatment and reduces the likelihood of re-hospitalization for congestive heart failure.
According to Edward Philbin, M.D., medical director of the Heart Failure Program and George Pataki Chair in Cardiology at Albany Medical Center, heart failure is characteristically accompanied by significant fluid retention. Aquapheresis is the first major advancement for acute fluid removal in the setting of acute heart failure since the introduction of diuretics, more than 50 years ago.
Aquapheresis uses the process of ultrafiltration to remove excess sodium and water from the body. Blood is withdrawn through a catheter and circulated through the filter system that separates the fluids from the blood. Once filtered, the clean blood is returned back to the body through a second catheter. Aquapheresis uses a peripheral intravenous line (a catheter inserted into a vein in the arm). By using this system, up to four liters of fluid can be removed in an eight-hour period, with no significant impact on blood pressure, kidney function, or electrolyte balance.
Conversely, treatment for congestive heart failure using intravenous or oral diuretic drugs can cause decreases in blood pressure and levels of potassium or magnesium, and may cause disruption of normal kidney function. Moreover, diuretics can take a longer period of time than aquapheresis to be completely effective. Overall, aquapheresis removes more fluid, more rapidly, than standard treatment with diuretics alone.
Even with severe disease, appropriate exercise can benefit those with CHF.1, 2 In a controlled trial, long-term (one year) exercise training led to improvements in quality of life and functional capacity in people with CHF.3 Nonetheless, too much exercise can be life-threatening for those with CHF. How much is “too much” varies from person to person; therefore, any exercise program undertaken by someone with CHF requires professional supervision.
Posts Tagged ‘Heart Failure’
Treatment of Congestive Heart Failure
07.10
Anemia Part I – Types of Anemia
06.26
It is hard to believe that America, a country with one of the highest standard living in the world having over 20 million people with the disease of anemia caused by unhealthy diet, and nutritional deficiency with protein and fat intake 30% more than any other country in the world. Most people understand that anemia is caused by iron deficiency in the bloodstream but in reality anemia is characterized by deficiency in the hemoglobin of the red blood cells diminishing the ability of the blood to transport oxygen to our cells and to removing carbon dioxide. In this article, we will discuss types of anemia.
1. Genetically passed through
Anemia is a genetic disease passing through from generation to generation.
a) Thalassmia
i) Thalassmia minor
It is the most common anemia in people of the Mediterranean descent. This is the mild case of anemia. The blood cell is smaller than normal blood cells and easily die off causing stress for the reproduction of new red blood cells because of not enough new blood cells to replace the loss of red blood cells.
ii) Thalassmia major
Thalassmia major is also called Cooley’s anemia. It is an inherited disorder that affects the production of normal hemoglobin. Thalassmia major is a deadly disease without frequent blood transfusions, people with this kind of anemia may not survive because of iron building up in heart and other organs, resulting in heart failure.
b) Aplastic anemia
This is one of the most deadly and rarest case of anemia. For whatever reason, the marrow bone that produces red blood cell does not work properly or it may be caused by an auto-immune disorder resulting in white blood cells attacking the bone marrow.
c) Sickle-celled anemia
This is a serious, life-threatening inherited form of anemia that mostly affects people of African ancestry. Sickle cell anemia is formed of hemoglobin deficiency causing red blood cells to become sticky, stiff, and more fragile resulting in the form of a curve and sickle-shape, restricting the circulation of blood in our body.
2. Deficiency of iron anemia
Iron is the most importance mineral in the blood, working together with zinc and copper to help to increase the oxygen level and circulation of our blood to nurture our body’s cells and removing carbon dioxide from the bloodstream. Iron is a hard to be absorbed mineral, without taking together with vitamin C, most iron in our body are wasted.
3 Deficiency of folate acid and vitamin B12 anemia (pernicious anemia)
Folate acid and vitamin B12 is vital for the reproduction of red blood cells caused by excessive drinking of alcohol and certain intake of certain medication such as oral contraceptive or anticancer drug. People eating only cooked foods have a highest risk of deficiency of folate and vitamin B12 anemia.
4. Chronic blood loss anemia
This type of anemia is resulted from a variety of chronic conditions such as hemorrhoids, cancer, menstruation and peptic ulcers.
5. Hemolytic anemia
In this case, the destruction of old red blood cells exceeds the production of new ones caused by the defective hemoglobin synthesis or trauma within the arteries, or caused by antibodies being produced by the immune system damaging red blood cells.

